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Independently verifiedCertified

CJC-1295 without DAC · 5mg

Purity (HPLC)>98%

LabVantage Analytical

Tested28 Jun 2026

Batch —·

Heavy metalsEndotoxinsMicrobialSolvents

View full COA

In stock

CJC-1295 without DAC

Name: CJC-1295 without DAC
Brand: AXOS
Availability: InStock

$69$13.80/mg

GHRH analog — pulsatile GH signaling research reagent

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Independently verifiedCertified

CJC-1295 without DAC · 5mg

Purity (HPLC)>98%

LabVantage Analytical

Tested28 Jun 2026

Batch —·

Heavy metalsEndotoxinsMicrobialSolvents

View full COA

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Researchers usually add these to complete the protocol.

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Certificate of analysis

Every vial ships with its own report

We don't publish a brand-wide spec sheet and call it a “COA.” The certificate you receive references the actual lot number stamped on your vial — with the raw chromatograms attached.

Lot-specific HPLC purity + LC-MS identity
Raw chromatogram + mass confirmation attached
Signed off by the lab's Principal Chemist
Permanent archive — searchable by lot ID

View all lab reports

Reference reportView All Live Reports

Vantage Analytical

Independent analytical lab

Certificate of Analysis

RESULT · PASS

ProductCJC-1295 without DAC · 5mg

Lot / batch—

Reported28 Jun 2026

MethodHPLC-UV + LC-MS

AppearanceWhite lyophilized powder

ClientSolace Peptides

Analytical results

Identity (LC-MS)1419.5 DaPASS

Purity (HPLC-UV)>98%PASS

Net content10.2 mgPASS

Endotoxin (LAL)< 0.05 EU/mgPASS

Heavy metals (ICP-MS)< 10 ppmPASS

Residual solventsBelow LOQPASS

HPLC Chromatogram

Purity >98%

Chemist verifiedDr. E. Hartley

Know your compound

What is CJC-1295 without DAC?

CJC-1295 without DAC (also known as Mod GRF 1-29) is a synthetic, truncated analog of growth hormone-releasing hormone (GHRH) that retains the first 29 amino acids of the native peptide. Unlike DAC-conjugated variants, this form lacks the drug affinity complex that extends plasma half-life, preserving the short-duration pharmacokinetic profile characteristic of endogenous GHRH.

The compound is studied because its brief activity window is thought to replicate the pulsatile character of physiological GHRH signaling — a feature DAC-extended analogs deliberately eliminate. Research applications center on GH-IGF-1 axis modulation, pituitary somatotroph responsiveness, and the consequences of maintaining versus disrupting natural GH pulse architecture in preclinical models.

Elevates GH and downstream IGF-1 in study models following administration
Preserves pulsatile GH release pattern, in contrast to long-acting GHRH analogs
Investigated for GH-IGF-1 axis modulation without sustained receptor occupancy

Lot-specific COAEvery lot ships with an HPLC Certificate of Analysis and mass-spectrometry identity confirmation.

Institutional fulfilmentShipped to verified institutional addresses with full analytical documentation.

Lot-specific COA: Every lot ships with an HPLC Certificate of Analysis and mass-spectrometry identity confirmation.
Institutional fulfilment: Shipped to verified institutional addresses with full analytical documentation.

For research use only — not for human or veterinary use.

The research

What the research actually found

The published findings behind this compound — the headline numbers, in plain terms, each cited to source. Preclinical results are directional (animal / in-vitro) and shown for context, not as therapeutic claims.

Published research on the reference compound class. Provided for scientific context only — not a claim about the product supplied.

Pulsatile GH patternPreclinical

A 2026 review describes CJC-1295 without DAC as clearing rapidly enough to produce discrete GH pulses — preserving the on-off rhythm of natural pituitary signaling — in contrast to DAC-conjugated analogs that sustain a flat elevation.

Frontiers in Endocrinology, 2026 · Review

Administration

GHRH analog delivered

GH pulse

Rapid somatotroph response

Clearance

Short half-life; receptor occupancy ends

Pulse resolved

Pulsatile pattern restored

Schematic only — no quantified half-life extractable from Dominikowski et al. 2026 without fabricating figures.

GH axis responsePreclinical

A 2026 narrative review by Dominikowski et al. characterizes GH elevation above baseline following GHRH analog administration in study models, with downstream IGF-1 rises reported as a secondary consequence — though the evidence base is mixed and no controlled magnitude or sample size was extractable.

Baseline GHbaseline

Post-administration GHelevated

Downstream IGF-1elevated

Directional only — bar heights are illustrative of reported direction, NOT measured magnitudes. No quantified values extractable from Dominikowski et al. 2026.

Frontiers in Endocrinology, 2026 · Review

CJC-1295 without DAC (5mg) — molecular specimen

GHRH receptor agonismpituitary somatotrophs

Pulsatile kineticsshort half-life profile

IGF-1 downstream inductionhepatic axis

Mechanism

How CJC-1295 without DAC works

CJC-1295 without DAC is studied to act through three distinct aspects of GHRH receptor biology, each reflecting a different dimension of its interaction with the GH-IGF-1 axis.

GHRH receptor agonismpituitary somatotrophs

The peptide is studied to bind and activate GHRH receptors on anterior pituitary somatotroph cells, stimulating GH synthesis and secretion in a manner analogous to endogenous GHRH.

Pulsatile kineticsshort half-life profile

Because it lacks a drug affinity complex, the compound clears rapidly, producing discrete GH secretory episodes rather than sustained receptor occupancy — a feature researchers use to study the physiological consequences of pulse-pattern GH signaling.

IGF-1 downstream inductionhepatic axis

GH released following receptor stimulation is studied to drive hepatic IGF-1 production, making this compound a tool for investigating the downstream somatotropic axis in preclinical and in vitro models.

For research use only. Mechanisms described reflect published preclinical and clinical literature.

The process

How every batch is verified

01
Synthesized
Produced under controlled manufacturing with documented, repeatable processes.
02
HPLC purity
An independent lab reports the exact purity of every lot — a number, never a 'passes'.
03
Mass-spec identity
Molecular weight confirms the exact sequence is what the label says.
04
COA published
The certificate is posted to the open library, by lot, before the vial ships.

Verified researchers

Reviewed by people who test it themselves

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FAQ

Frequently asked

Common questions about CJC-1295 without DAC

The COA guarantee

>98% pure — or your money back.

Have any lot independently re-tested. If the results don't match the published certificate, we refund the order in full — no returns, no questions.

Order with cover

Third-party tested

An independent, accredited lab — no commercial interest in the result.

A COA on every lot

A lot-specific certificate ships with each vial, archived by batch.

Test it yourself → refund

Re-test any batch at any lab. A mismatch is a full refund.

Every vial ships with its own report

We don't publish a brand-wide spec sheet and call it a “COA.” The certificate you receive references the actual lot number stamped on your vial — with the raw chromatograms attached.

Lot-specific HPLC purity + LC-MS identity

Raw chromatogram + mass confirmation attached

Signed off by the lab's Principal Chemist

Permanent archive — searchable by lot ID

View all lab reports

What is CJC-1295 without DAC?

Elevates GH and downstream IGF-1 in study models following administration

Preserves pulsatile GH release pattern, in contrast to long-acting GHRH analogs

Investigated for GH-IGF-1 axis modulation without sustained receptor occupancy

How CJC-1295 without DAC works

CJC-1295 without DAC is studied to act through three distinct aspects of GHRH receptor biology, each reflecting a different dimension of its interaction with the GH-IGF-1 axis.

GHRH receptor agonismpituitary somatotrophs

The peptide is studied to bind and activate GHRH receptors on anterior pituitary somatotroph cells, stimulating GH synthesis and secretion in a manner analogous to endogenous GHRH.

Pulsatile kineticsshort half-life profile

IGF-1 downstream inductionhepatic axis

For research use only. Mechanisms described reflect published preclinical and clinical literature.

Search

Search

Search

CJC-1295 without DAC

Every vial ships with its own report

What is CJC-1295 without DAC?

What the research actually found

How CJC-1295 without DAC works

GHRH receptor agonismpituitary somatotrophs

Pulsatile kineticsshort half-life profile

IGF-1 downstream inductionhepatic axis

How every batch is verified

Researchers reach for these

Frequently asked

What is CJC-1295 without DAC and how does it differ from CJC-1295 with DAC?

What has research shown about CJC-1295 without DAC and the GH-IGF-1 axis?

Is CJC-1295 without DAC FDA approved?

How is CJC-1295 without DAC used in research settings, and how does it compare to other GHRH analogs?

What is the molecular structure of CJC-1295 without DAC?

How should CJC-1295 without DAC be stored and handled?

>98% pure — or your money back.

Search

Search

CJC-1295 without DAC

Every vial ships with its own report

What is CJC-1295 without DAC?

What the research actually found

How CJC-1295 without DAC works

GHRH receptor agonismpituitary somatotrophs

Pulsatile kineticsshort half-life profile

IGF-1 downstream inductionhepatic axis

How every batch is verified

Researchers reach for these

Frequently asked

What is CJC-1295 without DAC and how does it differ from CJC-1295 with DAC?

What has research shown about CJC-1295 without DAC and the GH-IGF-1 axis?

Is CJC-1295 without DAC FDA approved?

How is CJC-1295 without DAC used in research settings, and how does it compare to other GHRH analogs?

What is the molecular structure of CJC-1295 without DAC?

How should CJC-1295 without DAC be stored and handled?

>98% pure — or your money back.