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Independently verifiedCertified

BPC-157 · 10mg

Purity (HPLC)≥98%

LabVantage Analytical

Tested28 Jun 2026

Batch —·

Heavy metalsEndotoxinsMicrobialSolvents

View full COA

In stock

BPC-157

Name: BPC-157
Brand: AXOS
Availability: InStock

$69$6.90/mg

Synthetic pentadecapeptide — tendon, gut, and vascular repair research

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Independently verifiedCertified

BPC-157 · 10mg

Purity (HPLC)≥98%

LabVantage Analytical

Tested28 Jun 2026

Batch —·

Heavy metalsEndotoxinsMicrobialSolvents

View full COA

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Certificate of analysis

Every vial ships with its own report

We don't publish a brand-wide spec sheet and call it a “COA.” The certificate you receive references the actual lot number stamped on your vial — with the raw chromatograms attached.

Lot-specific HPLC purity + LC-MS identity
Raw chromatogram + mass confirmation attached
Signed off by the lab's Principal Chemist
Permanent archive — searchable by lot ID

View all lab reports

Reference reportView All Live Reports

Vantage Analytical

Independent analytical lab

Certificate of Analysis

RESULT · PASS

ProductBPC-157 · 10mg

Lot / batch—

Reported28 Jun 2026

MethodHPLC-UV + LC-MS

AppearanceWhite lyophilized powder

ClientSolace Peptides

Analytical results

Identity (LC-MS)1419.5 DaPASS

Purity (HPLC-UV)≥98%PASS

Net content10.2 mgPASS

Endotoxin (LAL)< 0.05 EU/mgPASS

Heavy metals (ICP-MS)< 10 ppmPASS

Residual solventsBelow LOQPASS

HPLC Chromatogram

Purity ≥98%

Chemist verifiedDr. E. Hartley

Know your compound

What is BPC-157?

BPC-157 is a synthetic 15-amino-acid pentadecapeptide derived from a protective protein isolated from human gastric juice. It is among the most consistently studied compounds in preclinical regenerative biology, with a literature spanning tendon, gut, and vascular tissue models.

In rat Achilles transection models, BPC-157 accelerated tendon healing by 50–72% relative to controls — a magnitude that prompted sustained investigation into its pro-angiogenic and cytoprotective mechanisms. Parallel work in inflammatory bowel and wound models has extended this profile across multiple tissue compartments.

50–72% acceleration in tendon healing rates in rat transection models
Upregulates VEGFR2-driven angiogenesis across tendon, gut, and vascular tissue types
Promotes gut lining repair in preclinical inflammatory bowel models

Lot-specific COAEvery lot ships with an HPLC Certificate of Analysis and mass-spectrometry identity confirmation.

Institutional fulfilmentShipped to verified institutional addresses with full analytical documentation.

Lot-specific COA: Every lot ships with an HPLC Certificate of Analysis and mass-spectrometry identity confirmation.
Institutional fulfilment: Shipped to verified institutional addresses with full analytical documentation.

For research use only — not for human or veterinary use.

The research

What the research actually found

The published findings behind this compound — the headline numbers, in plain terms, each cited to source. Preclinical results are directional (animal / in-vitro) and shown for context, not as therapeutic claims.

Published research on the reference compound class. Provided for scientific context only — not a claim about the product supplied.

Tendon healing speedPreclinical

+50–72%

In a rat Achilles tendon transection model, BPC-157-treated animals healed 50–72% faster than untreated controls — the most quantified tissue-repair signal in the BPC-157 preclinical record.

vs. untreated controls · rat Achilles transection

Journal of Applied Physiology, 2011 · Chang et al.

BPC-157 treated+50–72% faster

Untreated controlBaseline rate

Bar height represents relative tendon-healing rate; control anchored to the midpoint of the observed acceleration range. Rat Achilles transection model.

Tendon cell migrationPreclinical

BPC-157 increased directed outgrowth and migration of tendon cells relative to controls in both an in vitro tendon cell assay and a rat Achilles transection model — suggesting a cellular mechanism behind the observed healing acceleration.

vs. untreated controls · in vitro + rat model

BPC-157Increased

ControlBaseline

Relative bar heights reflect directional difference only — no precise fold-change extracted from source. In vitro tendon cell outgrowth assay + rat Achilles transection model.

Journal of Applied Physiology, 2011 · Chang et al.

Angiogenic signallingPreclinical

Across gastrointestinal, tendon, ligament, muscle, and bone rodent models, BPC-157 consistently upregulated VEGFR2-associated angiogenic pathways — pointing to a tissue-agnostic pro-vascular mechanism reviewed across multiple independent studies.

in vitro · multiple rodent tissue models

Gut models

VEGFR2 upregulated · GI tract

Tendon & ligament

VEGFR2 upregulated · connective tissue

Muscle

VEGFR2 upregulated · skeletal muscle

Bone

VEGFR2 upregulated · bone repair

Timeline shows breadth of tissue compartments in which VEGFR2 upregulation was documented in rodent preclinical models. All findings are preclinical; no quantitative fold-change available.

Current Pharmaceutical Design, 2018 · Seiwerth et al.

AngiogenesisVEGFR2 upregulation

Nitric oxide modulationNO system interaction

Cell survival & migrationtendon / epithelial cells

Mechanism

How BPC-157 works

BPC-157 is studied to act through three distinct biological routes: pro-angiogenic receptor upregulation, nitric oxide system modulation, and direct cytoprotective effects on tendon and epithelial cells.

AngiogenesisVEGFR2 upregulation

Preclinical models indicate BPC-157 upregulates VEGFR2, the primary receptor driving new blood vessel formation, thereby increasing vascular ingrowth into injured tissue compartments.

Nitric oxide modulationNO system interaction

Studies in rat models suggest BPC-157 interacts with the nitric oxide signalling pathway at wound sites, modulating vasoactive and cytoprotective responses independent of its angiogenic effects.

Cell survival & migrationtendon / epithelial cells

In vitro and rat transection data indicate BPC-157 promotes tendon cell outgrowth, survival under stress conditions, and directed cell migration — mechanisms studied as contributors to structural tissue repair.

For research use only. Mechanisms described reflect published preclinical and clinical literature.

The process

How every batch is verified

01
Synthesized
Produced under controlled manufacturing with documented, repeatable processes.
02
HPLC purity
An independent lab reports the exact purity of every lot — a number, never a 'passes'.
03
Mass-spec identity
Molecular weight confirms the exact sequence is what the label says.
04
COA published
The certificate is posted to the open library, by lot, before the vial ships.

Verified researchers

Reviewed by people who test it themselves

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FAQ

Frequently asked

Common questions about BPC-157

The COA guarantee

≥98% pure — or your money back.

Have any lot independently re-tested. If the results don't match the published certificate, we refund the order in full — no returns, no questions.

Order with cover

Third-party tested

An independent, accredited lab — no commercial interest in the result.

A COA on every lot

A lot-specific certificate ships with each vial, archived by batch.

Test it yourself → refund

Re-test any batch at any lab. A mismatch is a full refund.

Every vial ships with its own report

We don't publish a brand-wide spec sheet and call it a “COA.” The certificate you receive references the actual lot number stamped on your vial — with the raw chromatograms attached.

Lot-specific HPLC purity + LC-MS identity

Raw chromatogram + mass confirmation attached

Signed off by the lab's Principal Chemist

Permanent archive — searchable by lot ID

View all lab reports

What is BPC-157?

50–72% acceleration in tendon healing rates in rat transection models

Upregulates VEGFR2-driven angiogenesis across tendon, gut, and vascular tissue types

Promotes gut lining repair in preclinical inflammatory bowel models

How BPC-157 works

AngiogenesisVEGFR2 upregulation

Preclinical models indicate BPC-157 upregulates VEGFR2, the primary receptor driving new blood vessel formation, thereby increasing vascular ingrowth into injured tissue compartments.

Nitric oxide modulationNO system interaction

Studies in rat models suggest BPC-157 interacts with the nitric oxide signalling pathway at wound sites, modulating vasoactive and cytoprotective responses independent of its angiogenic effects.

Cell survival & migrationtendon / epithelial cells

For research use only. Mechanisms described reflect published preclinical and clinical literature.

Search

Search

Search

BPC-157

Every vial ships with its own report

What is BPC-157?

What the research actually found

How BPC-157 works

AngiogenesisVEGFR2 upregulation

Nitric oxide modulationNO system interaction

Cell survival & migrationtendon / epithelial cells

How every batch is verified

Researchers reach for these

Frequently asked

What is BPC-157 and where does it originate?

What has preclinical research shown about BPC-157's activity?

Is BPC-157 FDA approved?

How is BPC-157 used in research settings, and how does it differ from standard angiogenic growth factors?

What is the molecular structure of BPC-157?

How should BPC-157 research stock be stored?

≥98% pure — or your money back.

Search

Search

BPC-157

Every vial ships with its own report

What is BPC-157?

What the research actually found

How BPC-157 works

AngiogenesisVEGFR2 upregulation

Nitric oxide modulationNO system interaction

Cell survival & migrationtendon / epithelial cells

How every batch is verified

Researchers reach for these

Frequently asked

What is BPC-157 and where does it originate?

What has preclinical research shown about BPC-157's activity?

Is BPC-157 FDA approved?

How is BPC-157 used in research settings, and how does it differ from standard angiogenic growth factors?

What is the molecular structure of BPC-157?

How should BPC-157 research stock be stored?

≥98% pure — or your money back.